Results: Positive or Anti-atherogenic results:
Total LDL dropped from 111 to 106 mg/dl.
IDL dropped from 17 to 6 mg/dl.
HDL increased from 60 to 76 mg/dl.
HDL2 increased from 17 to 24 mg/dl.
HDL3 increased from 43 to 52 mg/dl.
Total VLDL decreased from 22 to 18 mg/dl.
VLDL3 decreased from 13 to 11 mg/dl mg/dl.
Triglycerides decreased from 100 to 66 mg/dl.
Non-HDL cholesterol decreased from 133 to 124 mg/dl.
Remnant lipoproteins IDL+VLD3 decreased from 30 to 19mg/dl.
Testosterone increased from 586 to 841 ng/dl.
Results: Negative or potentially atherogenic:
Total Cholesterol increased from 192 to 200 mg/dl.
Lipoprotein A increased from 6 to 8 mg/dl.
In summary, the ONLY negative blood marker found could be lipoprotein A, which is one of the "bad-bad" LDL cholesterols increased from 6-8 mg/dl. Total cholesterol did increase by 8 mg/dl but the overall picture of total cholesterol was by all accounts greatly improved. Most significantly, Testosterone increased ~70%, from 586-841 and triglycerides decreased 34%, dropping from 100 to 66. HDL also increased ~27%, from 60 to 76.
Trigylcerides to HDL ("the good cholesterol") ratio has statistically shown to be one of the most potent predictors of heart disease (17, 18), and also all cause mortality (19). A Harvard study found that people with the highest ratio of triglycerides to HDL had 16x the risk of heart attack as those with the lowest ratio. Furthermore, high triglycerides alone increased the risk by 3x. Triglycerides/HDL was found to be a better predictor of heart disease than HDL/LDL and certainly total cholesterol. So in terms of the triglyceride to HDL ratio:
- 2 or less is considered ideal
- 2-4 is at risk
- 4-6 high risk
- 6+ plan a funeral
"If you eat too much food containing cholesterol and/or saturated fat, the level of cholesterol in your blood will rise. The excess cholesterol will be deposited in artery walls, causing them to thicken and narrow. In time this will block blood supply to the heart (and other organs) causing a heart attack or stroke."
- The inflamed cells cannot make normal amounts of anti-thrombotic and vasodilating cytokines.
- Numerous inflammatory cytokines are released.
- Macrophages adhere to injured endothelium.
- These macrophages then release enzymes and toxic oxygen radicals (that oxidize LDL) and further damage the injured vessel.
- Growth factors are released which stimulate smooth muscle cell proliferation in the affected vessel. (4)
- Our ratio of omega 6 to omega 3 is skewed in a pro-inflammatory way. Evolution prescribed a 1:1 to 3:1 ratio of these fatty acids while our SAD diets typically are in the range of 15:1 to 16.7:1 (5). Negative effects of this ratio include inflammation (which progresses a plethora of disease states), oxidization of LDL and inhibition of the anti-inflammatory benefits of omega 3 fatty acids.
- Our highly refined carbohydrate diets lead to inflammation via elevated blood glucose and insulin resistance(6).
- Food allergies and intolerances often from relatively novel such foods as dairy (lactose) and wheat (gluten) can elevate inflammation in various tissues in the body. (7,8).